Focal paroxysmal kinesigenic choreoathetosis.
نویسنده
چکیده
Three cases of paroxysmal kinesigenic choreoathetosis are described in whom unilateral attacks were focally induced, together with a case in whom bilateral attacks only occured. Treatment with phenytoin was effective in all cases. The aspects of the literature relating to focal and generalised attacks in paroxysmal kinesigenic choreoathetosis are reviewed.
منابع مشابه
Paroxysmal kinesigenic choreoathetosis in hyperthyroidism.
Paroxysmal kinesigenic choreoathetosis is an unusual movement disorder often triggered by attempts to use the limbs, and has sometimes been associated with diffuse or focal brain injury. We report its occurrence in hyperthyroidism, with which choreoathetosis has rarely been described in the past without known cause. Choreoathetosis has also occurred with other metabolic and toxic disorders, and...
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A case of paroxysmal kinesigenic choreoathetosis is described. A hitherto unreported response to l-dopa is recorded and its possible relevance to the pathophysiology of the disorder is discussed.
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Paroxysmal kinesigenic dyskinesia (PKD) is presented as a short paroxysmal attack of focal or generalized involuntary movement. Bilateral striopallidodentate calcinosis (BSPDC) is referred to as Fahr's disease and is characterized by the calcification of the basal ganglia, cerebellar nuclei and thalamus. Most common presentation of BSPDC was a parkinsonism, and PKD has also been reported to few...
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Epilepsy and paroxysmal dyskinesia are two episodic cerebral disorders that can share a common genetic basis. Rare families with infantile seizures and paroxysmal dyskinesia [predominantly paroxysmal kinesigenic dyskinesia (PKD)], co-inherited as a single autosomal dominant trait, have been described (infantile convulsions with paroxysmal choreoathetosis; ICCA syndrome) and a disease gene has b...
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A [(18)F]-FDG PET study was performed in a 44 year old man with proximal kinesigenic choreoathetosis (PKC) secondary to idiopathic primary hypoparathyroidism (IPH) before and 1 year after calcium/calcitriol therapy. The [(18)F]-FDG PET performed before the start of the therapy disclosed a significant bilateral hypometabolism in the ventral striatum. One year later, with the patient still under ...
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عنوان ژورنال:
- Journal of neurology, neurosurgery, and psychiatry
دوره 46 4 شماره
صفحات -
تاریخ انتشار 1983